打字猴:1.70000291e+09
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1700002913 鸟类中的霍尔丹法则
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1700002915 Hill GE, Johnson JD. The mitonuclear compatibility hypothesis of sexual selection. Proceedings Royal Society B280: 20131314 (2013).
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1700002917 Mittwoch U. Phenotypic manifestations during the development of the dominant and default gonads in mammals and birds. Journal of Experimental Zoology281: 466–71 (1998).
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1700002921 Suarez RK. Oxygen and the upper limits to animal design and performance. Journal of Experimental Biology201: 1065–72 (1998).
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1700002923 触发细胞凋亡的死亡门槛
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1700002925 Lane N. Bioenergetic constraints on the evolution of complex life. Cold Spring Harbor Perspectives in Biology. doi: 10.1101/cshperspect.a015982 (2014).
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1700002927 Lane N. The costs of breathing. Science334: 184–85 (2011).
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1700002929 人类早期隐性流产的发生率
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1700002935 自由基老化理论
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1700002937 Barja G. Updating the mitochondrial free-radical theory of aging: an integrated view, key aspects, and confounding concepts. Antioxidants and Redox Signalling19: 1420–45 (2013).
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1700002941 Harmann D. Aging – a theory based on free-radical and radiation chemistry. Journal of Gerontology11: 298–300 (1956).
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1700002943 Murphy MP. How mitochondria produce reactive oxygen species. Biochemical Journal417: 1–13 (2009).
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1700002945 自由基老化理论的问题
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1700002947 Bjelakovic G, Nikolova D, Gluud LL, Simonetti RG, Gluud C. Antioxidant supplements for prevention of mortality in healthy participants and patients with various diseases. Cochrane Database of Systematic Reviewsdoi: 10.1002/14651858.CD007176 (2008).
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1700002949 Gutteridge JMC, Halliwell B. Antioxidants: Molecules, medicines, and myths. Biochemical Biophysical Research Communications393: 561–64 (2010).
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1700002953 Moyer MW. The myth of antioxidants. Scientific American308: 62–67 (2013).
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1700002955 自由基信号与衰老的关系
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1700002957 Lane N. Mitonuclear match: optimizing fitness and fertility over generations drives ageing within generations. BioEssays33: 860–69 (2011).
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1700002959 Moreno-Loshuertos R, Acin-Perez R, Fernandez-Silva P, Movilla N, Perez-Martos A, de Cordoba SR, Gallardo ME, Enriquez JA. Differences in reactive oxygen species production explain the phenotypes associated with common mouse mitochondrial DNA variants. Nature Genetics38: 1261–68 (2006).
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