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[5] W einberg, R. (1998). One renegade cell. Weidenfeld and Nicolson,London.
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[6] L evine, A. J. (1997). p53, the cellular gatekeeper for growth and division. Cell 89: 323-31.
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[7] L owe, S. W. (1995). Cancer therapy and p53. Current Opinion in Oncology 7: 547-53.
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[8] H ueber, A. O. and Evan, G. I. (1998). Traps to catch unwary oncogenes.Trends in Genetics 14: 364-7.
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[9] C ook-Deegan, R. (1994). The gene wars: science, politics and the human genome. W. W. Norton, New York.
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[10] Krakauer, D. C. and Payne, R. J. H. (1997). The evolution of virusinduced apoptosis. Proceedings of the Royal Society of London, Series B 264: 1757-62.
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[11] LeGrand, E. K. (1997). An adaptationist view of apoptosis. Quarterly Review of Biology 72: 135-47.
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18号染色体
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杰夫·莱昂（Geoff Lyon）和彼得·戈梅尔（Peter Gomel）的《改变命运》（美国诺顿出版社，1996）一书对基因疗法的发展做了详尽的论述，起了个很不错的头。斯蒂芬·诺丁汉（Stephen Nottingham）的《吃掉你的基因》（泽德出版社，1998）详述了植物基因工程的历史。李·西尔弗（Lee Silver）的《复制之谜：性、遗传和基因再造》（魏登菲尔德·尼科尔森出版社，1997）探讨了生殖技术和基因工程对人类的影响。
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[1] V erma, I. M. and Somia, N. (1997). Gene therapy — promises, problems and prospects. Nature 389: 239-42.
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[2] C arter, M. H. (1996). Pioneer Hi-Bred: testing for gene transfers. Harvard Business School Case Study N9-597-055.
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[3] C ap e c chi, M. R. (1989). Alt ering the g enome by homolo gou s recombination. Science 244: 1288-92.
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[4] First, N. and Thomson, J. (1998). From cows stem therapies? Nature Biotechnology 16: 620-21.
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19号染色体
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许多书刊报中都对基因筛查的利弊进行了详尽的讨论，但却缺乏让人眼睛为之一亮的作品。钱德勒·伯尔（Chandler Burr）的《再创：生物学如何使我们成为同性恋》（矮脚鸡出版社，1996）正是难得的精品。
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[1] L yon, J. and Gorner, P. (1996). Altered fates. Norton, New York.
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[2] E to, M., Watanabe, K. and Makino, I. (1989). Increased frequencies of apolipoprotein E2 and E4 alleles in patients with ischemic heart disease.Clinical Genetics 36: 183-8.
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[3] L ucotte, G., Loirat, F. and Hazout, S. (1997). Patterns of gradient of apolipoprotein E allele *4 frequencies in western Europe. Human Biology 69: 253-62.
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[4] Kamboh, M. I. (1995). Apolipoprotein E polymorphism and susceptibility to Akheimer’s disease. Human Biology 67: 195-215; Flannery, T. (1998).Throwim way leg. Weidenfeld and Nicolson, London.
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[5] C ook-Degan, R. (1995). The gene wars: science, politics and the human genome. Norton, New York.
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[6] Kamboh, M. I. (1995). Apolipoprotein E polymorphism and susceptibility to Alzheimer’s disease. Human Biology 67: 195-215; Corder, E. H. et al.(1994). Protective effect of apolipoprotein E type 2 allele for late onset Alzheimer disease. Nature Genetics 7: 180-84.
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[7] B ickeboller, H. et al. (1997). Apolipoprotein E and Alzheimer disease
:genotypic-specific risks by age and sex. American Journal of Human Genetics 60: 439-46; Payami, H. et al. (1996). Gender difference in apolipoprotein E-associated risk for familial Alzheimer disease: a possible clue to the higher incidence of Alzheimer disease in women.American Journal of Human Genetics 58: 803-11; Tang, M. X. et al.(1996). Relative risk of Alzheimer disease and age-at-onset distributions,based on APOE genotypes among elderly African Americans, Caucasians and Hispanics in New York City. American Journal of Human Genetics 58: 574-84.
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[8] C aldicott, F. et al. (1998). Mental disorders and genetics: the ethical context. Nuffield Council on Bioethics, London.
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[9] B ickeboller, H. et al. (1997). Apolipoprotein E and Alzheimer disease
:genotypic-specific risks by age and sex. American Journal of Human Genetics 60: 439-46.
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[10] M addox, J. (1998). What remains to be discovered. Macmillan, London.
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